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Deficient Pms2, ERCC1, Ku86, CcOI in Field Defects During Progression to Colon Cancer
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July 28, 2010
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1Department of Cell Biology and Anatomy, College of Medicine,University of Arizona, Tucson, 2Southern Arizona Veterans Affairs Health Care System,Tucson, AZ, 3Department of Surgery, College of Medicine,University of Arizona, Tucson, 4Biomedical Diagnostics and Research,Tucson, AZ, 5Department of Medicine, College of Medicine,University of Arizona, Tucson
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Deficient Pms2Deficient ERCC1Deficient Ku86Deficient CcOIField DefectColon CancerCarcinogenesisMolecular AbnormalitiesColonic AdenocarcinomaAdenoma With Advanced NeoplasiaImmunohistochemistryDNA Repair ProteinsApoptosisDNA Mismatch RepairDNA Nucleotide Excision RepairApoptosis ResistanceMutabilityProgression To Colon CancerDNA Non-homologous End JoiningCytochrome C Oxidase Subunit
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Nguyen, H., Loustaunau, C., Facista, A., Ramsey, L., Hassounah, N., Taylor, H., Krouse, R., Payne, C. M., Tsikitis, V. L., Goldschmid, S., Banerjee, B., Perini, R. F., Bernstein, C. Deficient Pms2, ERCC1, Ku86, CcOI in Field Defects During Progression to Colon Cancer. J. Vis. Exp. (41), e1931, doi:10.3791/1931 (2010).
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