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1Department of Cell Biology and Anatomy, College of Medicine,University of Arizona, Tucson, 2Southern Arizona Veterans Affairs Health Care System,Tucson, AZ, 3Department of Surgery, College of Medicine,University of Arizona, Tucson, 4Biomedical Diagnostics and Research,Tucson, AZ, 5Department of Medicine, College of Medicine,University of Arizona, Tucson
Kapitel
- 00:05Titel
- 01:47Introduction
- 03:04Preparing Tissues
- 04:15Immunostaining
- 08:16Evaluating Tissue Sections
- 27:11Conclusion
Pms2 ve / veya bütün kript olarak ERCC1 Küçültülmüş / eksik ifade kolon adenokarsinomların, muhtemelen yüksek değişkenliktir ve kanser ilerleme alanı defekti temelinde her iki tarafında 10 cm içinde sık rastlanan bir olaydır. Ku86 veya CcOI eksikliği, bu alanı defektleri çok daha az sık.
Tags
Deficient Pms2Deficient ERCC1Deficient Ku86Deficient CcOIField DefectColon CancerCarcinogenesisMolecular AbnormalitiesColonic AdenocarcinomaAdenoma With Advanced NeoplasiaImmunohistochemistryDNA Repair ProteinsApoptosisDNA Mismatch RepairDNA Nucleotide Excision RepairApoptosis ResistanceMutabilityProgression To Colon CancerDNA Non-homologous End JoiningCytochrome C Oxidase Subunit
