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1Department of Cell Biology and Anatomy, College of Medicine,University of Arizona, Tucson, 2Southern Arizona Veterans Affairs Health Care System,Tucson, AZ, 3Department of Surgery, College of Medicine,University of Arizona, Tucson, 4Biomedical Diagnostics and Research,Tucson, AZ, 5Department of Medicine, College of Medicine,University of Arizona, Tucson
Kapitel
- 00:05Titel
- 01:47Introduction
- 03:04Preparing Tissues
- 04:15Immunostaining
- 08:16Evaluating Tissue Sections
- 27:11Conclusion
Уменьшение / отсутствует выражение Pms2 и / или ERCC1 во всем склепов является частым событием в пределах 10 см с каждой стороны толстой аденокарциномы, вероятно основе полей дефектов с высокой изменчивости и прогрессии рака. Недостаток Ku86 или CcOI гораздо реже в этих полей дефектов.
Tags
Deficient Pms2Deficient ERCC1Deficient Ku86Deficient CcOIField DefectColon CancerCarcinogenesisMolecular AbnormalitiesColonic AdenocarcinomaAdenoma With Advanced NeoplasiaImmunohistochemistryDNA Repair ProteinsApoptosisDNA Mismatch RepairDNA Nucleotide Excision RepairApoptosis ResistanceMutabilityProgression To Colon CancerDNA Non-homologous End JoiningCytochrome C Oxidase Subunit
